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Osteonecrosis (avascular necrosis / AVN)
C/P
- Early stage asymptomatic — usually advanced by presentation
- Pain (commonest)
- Clicking (loose articular fragment snapping)
Inves
- X-ray normal in early disease
- Crescent sign — thin radiolucent crescent below convex articular surface — earliest X-ray sign
- MRI = gold standard
- Bone scan if MRI unavailable
Mng
- Conservative — bed rest, non-weight bearing, analgesia, treat underlying cause
- Surgical — core decompression, osteotomy, replacement
Special
- Sites: femoral head, distal femur, scaphoid, proximal humerus, talus, MT (Freiberg's), navicular (Kohler's), lunate (Kienbock's)
- Non-traumatic: infection, sickle cell, Gaucher's (lipid storage → fat emboli), Caisson (decompression), thrombophilia, steroids, alcohol, ionising radiation
- Traumatic AVN — interruption of blood supply by displaced fractures (hip, scaphoid, talus)
physiology · background · low-yield
Mechanism
- Traumatic AVN also via thrombosis of intravascular capillaries in un-displaced fractures (not only vessel interruption in displaced #)
Background
- Femoral-head blood supply — lateral + medial circumflex branches of profunda femoris → retinacular vessels at the base of the femoral neck (intra-capsular)